Well, folks, another piece to the health puzzle has fallen into place, Bless God’s Helpers. I always pray for understanding what I don’t understand and this was a BIG piece of the puzzle. I’ve had what I suspected was Lyme disease, but as I researched it, I realized that it was not a disease, but likely a parasitic infection brought on not by ticks as the media misled people to believe, but the more likely culprit was the sudden epidemic of bed bugs, which had not been a problem since the 1970s. My blog on Lyme here.
Staying in my van, I have to use a potty bucket for night and when no public toilet is available.
And I have been given the not-so-subtle hint that I stink. I knew that I wasn’t as fresh as I would be having a shower, but I sponge bathe every night so I cleanse myself regularly.
But something happened to open my eyes to something else going on. Without being too graphic, my urine smelled very strong of ammonia. I researched it online, and every single webpage just had a pat answer that it was just dehydration.
I knew that I wasn’t dehydrated, so I continued to dig for answers.
I learned that strong ammonia in urine and feces could mean liver failure (the gut normally has ammonia, but the liver is supposed to cleanse that, so if you smell of ammonia, your liver is not functioning properly), kidney failure, and neurotoxicity (poisoning the brain).
And because I have repeatedly seen parasites in my coffee enemas, I wondered if parasites emitted ammonia as a waste disposal function?
Yep. There is a parasite that emits ammonia as it dies. And not only that, it can cause cerebral malaria (CM).
Here’s a scientific article on the parasite and it’s causing terrible harm with high ammonia levels in the body as well as brain. It can even affect one part of the brain in concentrated amounts.
From the article:
Cerebral malaria (CM) is the most severe complication associated with Plasmodium falciparum infection. The exact pathogenic mechanisms leading to the development of CM remains poorly understood while the mortality rates remain high. Several potential mechanisms including mechanical obstruction of brain microvasculature, inflammation, oxidative stress, cerebral energy defects, and hemostatic dysfunction have been suggested to play a role in CM pathogenesis. However, these proposed mechanisms, even when considered together, do not fully explain the pathogenesis and clinicopathological features of human CM. This necessitates consideration of alternative pathogenic mechanisms. P. falciparum generates substantial amounts of ammonia as a catabolic by-product, but lacks detoxification mechanisms. Whether this parasite-derived ammonia plays a pathogenic role in CM is presently unknown, despite its potential to cause localized brain ammonia elevation and subsequent neurotoxic effects. This article therefore, explores and proposes a potential role of parasite-derived ammonia in the pathogenesis and neuropathology of CM. A consideration of parasite-derived ammonia as a factor in CM pathogenesis provides plausible explanations of the various features observed in CM patients including how a largely intravascular parasite can cause neuronal dysfunction.
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Cerebral malaria (CM) is one of the most common and fatal complications associated with Plasmodium falciparum infection (Newton et al., 2000). Despite the availability of effective anti-malarial drugs, CM mortality rates in malaria afflicted regions remain high, ranging between 15 and 20% (Mishra and Newton, 2009). This has partly been attributed to the fact that a number of patients succumb to CM before the onset of administered anti-malarial therapeutic effects (Mishra and Newton, 2009). Transient and persistent neurocognitive impairments are also common among cerebral malaria survivors, mostly children (van der Wal et al., 2005; Idro et al., 2010). Several mechanisms have been hypothesized to play a role in the pathogenesis of CM including; mechanical obstruction of brain microvasculature by sequestered parasitized red blood cells (PRBCs), inflammation, hemostatic dysfunction, excessive parasite-derived lactate, and oxidative stress (Berendt et al., 1994; Medana et al., 2001; van der Heyde et al., 2006; Mariga et al., 2014). However, the exact pathogenesis of CM remains poorly understood and the various clinicopahtological features of human CM cannot be fully explained by the existing hypotheses (van der Heyde et al., 2006). Additionally, adjunctive therapies targeting some of these earlier proposed pathogenic mechanisms have failed to improve CM treatment outcomes or have been found detrimental in human clinical studies (White et al., 2013). Exploration of other potential pathogenic factors is therefore needed in order to understand the underlying CM pathogenesis and develop adjunctive therapies that can possibly prolong anti-malarial therapeutic window and reduce neurocognitive sequelae among survivors.
Ammonia is a neurotoxic metabolic by-product whose blood concentration is tightly maintained at levels of less than 50 μmol/L mainly by the action of liver glutamine synthetase, which has a lower Km for ammonia than carbamoyl-phosphate synthetase (Arn et al., 1990; Cohn and Roth, 2004; Adeva et al., 2012). The brain astrocytic glutamine synthetase also plays a minor role, converting upto 25% of blood derived ammonia to glutamine (Eid and Lee, 2013). Several factors including, acute liver failure (ALF), urea cycle disorders, Reye’s syndrome and hepatic drug toxity may however interfere with the normal ammonia metabolism leading to hyperammonemia (Cohn and Roth, 2004). Regardless of the cause, elevations of blood ammonia concentration above the normal physiological levels often lead to encephalopathies (Haberle, 2013). Interestingly, the clinicopathological features of acquired and congenital hyperammonemic encephalopathies such as coma, brain edema, and seizures (Summar et al., 2008; Haberle, 2013) are analogous to some of the features observed in CM patients (Idro et al., 2005). Moreover, experimental cerebral malaria (ECM) in murine models has also revealed metabolic changes that point toward parasite-induced perturbation of ammonia detoxification (Ghosh et al., 2012). These observations, coupled to the fact that P. falciparum generates large amounts of ammonia in the absence of intrinsic parasite detoxification mechanisms (Zeuthen et al., 2006) clearly suggests a potential role of parasite-derived ammonia in CM pathogenesis.
It was recently hypothesized that development of ALF in malaria patients may lead to accumulation of ammonia and other neurotoxins, which then enter the brain aided by blood brain barrier (BBB) breakdown and ultimately cause neurological damage and manifestation of CM (Martins and Daniel-Ribeiro, 2013). However, several studies indicates that parasite-induced liver dysfunction is not a common occurrence in P. falciparum malaria (Anand et al., 1992; Murthy et al., 1998; Nacher et al., 2001; Mazumder et al., 2002; Mohanty et al., 2004; Prommano et al., 2005; Kochar et al., 2010; Whitten et al., 2011). Therefore, it is conceivable that sequestered parasites in the brain vasculature known to produce large amounts of ammonia as a catabolic by-product, may disrupt normal brain ammonia metabolism leading to local accumulation of ammonia. This possibility has however not been considered as a pathogenic factor in CM. The aim of the current article is therefore to explore potential mechanisms by which P. falciparum may cause localized elevation of brain ammonia and subsequent neurotoxicity.
Potential ammonia-glutamine vicious cycle in which intravascular malaria parasites catabolize glutamine, releasing ammonia, which is taken up into the astrocyte via transporters including NCCK1 and conjugated with glutamate (derived from α-ketoglutarate) to form glutamine in a glutamine synthetase (GS) catalyzed reaction. The generated glutamine may then be released back to blood thus resupplying the parasite with the necessary glutamine. The GS catalyzed reaction also plays a key role in the glutamine-glutamate cycle, in which released synaptic glutamate is recycled back to the glutamatergic neurons in form of glutamine and converted by glutaminase to form glutamate neurotransmitter.
So this is one more piece to the affliction that isn’t Lyme disease, but again, a parasitic infection.
The CDC has recently stated that Malaria was on the rise in the United States….but stopped short of telling the public the REAL reason it was on the uptick!! That is outrageous and proves a lack of ethics to inform the American public about the whole truth of what is happening. The public will dismiss it if they have not traveled outside the United States, not even realizing they can be infected right here!
Somehow we are being assaulted with parasites. My guess is the bed bugs, as I stated earlier, but it could also be in our food, water, and even sprayed on us. If you recall, I blogged on the disappearing crickets. I suddenly stopped hearing crickets and found out that they were being destroyed by horsehair parasites that takeover their bodies and destroy them within.
I do believe covid was also parasitic infection. If you recall, ivermectin, a drug to kill parasites, was being recommended to battle covid. That didn’t make any sense at the time because the lying media kept saying it was a virus. Ivermectin killed parasites, not viruses.
When I discovered this yesterday, Hillary Clinton popped into my head. She was livid that I had made the connection. Later, Bill Clinton, a known secret society member of the Knights of Malta, also popped into my head, angry. That, to me, clinches it that they were involved in this bioweapon in some way.
I thought about the researcher in an East coast lab that was targeted and killed (committed suicide?) after his involvement with an experimental bioweapon. I’m trying to jog my memory, but when I research it, I keep getting that horrible show that glorifies drugs. Not going to name it nor link to it.
Here’s an interesting page on the history of bioweapons:
Table 1
Examples of biological warfare during the past millennium
| Year | Event |
|---|---|
| 1155 | Emperor Barbarossa poisons water wells with human bodies, Tortona, Italy |
| 1346 | Mongols catapult bodies of plague victims over the city walls of Caffa, Crimean Peninsula |
| 1495 | Spanish mix wine with blood of leprosy patients to sell to their French foes, Naples, Italy |
| 1650 | Polish fire saliva from rabid dogs towards their enemies |
| 1675 | First deal between German and French forces not to use ‘poison bullets’ |
| 1763 | British distribute blankets from smallpox patients to native Americans |
| 1797 | Napoleon floods the plains around Mantua, Italy, to enhance the spread of malaria |
| 1863 | Confederates sell clothing from yellow fever and smallpox patients to Union troops, USA |
The last one of selling clothing with disease to the Union troops is a new one for me.
#$%&* Confederates!!
So, bioweapons have been used against people for a long, long time. If you believe the United States is under attack, then it is entirely possible that bioweapons are being used. A sneaky weapon for a sneaky war….
Quinine is one of the treatments for malaria. I recently started drinking tonic water with quinine in it.
And guess what has started happening? Drug stores that stocked quinine water have stopped stocking it. And worse, the stores that stock quinine water have a poor quality with sugar or sugar substitute in it. Unbelievable.
What to do about parasites and ammonia? Well, there are several natural herbs that are recommended. I’ve tried them without much success. However, I started thinking about ammonia smell being neutralized by vinegar and baking soda….hmmm….and vinegar kills most parasites….these may be a treatment, but I don’t know for sure. I just know that trying Ivermectin and wormwood and cloves have killed some, but not all.
*Edited to add: so I tried vinegar, and it helped my low grade bladder infection, but then I started noticing my hair getting thinner and even more alarming, the spot where I broke my leg in 2019 was very swollen. I mean seriously swollen. It looked as bad as if I had just broken it.
Then I remembered — there was a recipe in a book on boiled eggs being placed in vinegar to remove the shell. The vinegar dissolved calcium! Shoot, that was why I was seeing issues with my poor bones, and likely the reason for my hair suddenly getting thinner. Fudge. Back to the drawing board, as they say.
And I’m sure the Clintons know what the cure is for these horrible parasites. Something they did in their administration. f-ing satanists!!
UPDATE: I found a couple of interesting webpages regarding the introduction of malaria as a bioweapon by the Nazis. The time that Nazis used mosquitoes as biological weapon and evidence for biological weapons of the Third Reich.
Since the Rothschilds (satanists) are using the same playbook they used for the Nazis and World War II, my instincts are that the Clintons (satanists) unleashed this bioweapon amongst those of us who won’t take the mark of the beast. And even those whom have joined. I do believe this is across the United States, since the CDC has admitted an uptick in malaria.
But in case you have joined the cult, thinking that you were “safe” and that they would “take care of you”, well let me open your eyes.
They are ruthless. They consider everyone who is not a part of the Dark Nobility as “useless eaters”. Hillary Clinton has been quoted as saying just that. You may think you are protected because you joined the Cult, because they tell you that they are protecting you from the evil ones ( those who aren’t part of the cult, ironically), but let me tell you that if you are being besieged, it is THEM doing it!!
I cannot stress that enough. I know THE CULT is targeting those members who don’t really get it. They are Good Hearts being kind, compassionate, unselfish, and so on, and don’t know about the evil works done behind their backs. If you have experienced many flat tires in the last five years or so, or car problems, work problems, or marriage problems, you need to really open your eyes up to those who are working to cause these issues.
I know that the Cult messed with marriages since at least World War I, and probably even longer. I believe they had cult members marry non-cult members to try to get them to sell their soul to the evil one.
And if you have had flat tires, but then a cult members shows up and acts all kind and helpful, be aware that the cult probably sent someone to stick something in the tire. They have even used Directed Energy (DEW) to flatten tires and I think to break windows. I have seen a LOT of broken windows on gangstalker vehicles. I suspect they refused to go along with it and were punished.
Eagle 